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In addition, the observed differential 1-MNA steady-state levels following NNMT inhibition may reflect differential transcriptional regulation of cellular metabolic pathways across the various stages of 3T3-L1 differentiation;[38] for example, aldehyde oxidase (AOX)-mediated degradation of 1-MNA may be more pronounced in pre-adipocytes compared to adipocytes, thereby synergistically reducing 1-MNA levels to a greater extent in pre-adipocytes compared to adipocytes

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Further research is needed to elucidate the specific roles of individual members of the AKR family in different cancer types, identify biomarkers of AKR activity, and optimize therapeutic approaches to effectively target AKRs in cancer treatment